Congratulations Ahmed A. Abood, M.D., PhD
The assessment committee consisted of one Chairman and two Opponents:
Chairman:
Torben Bæk Hansen, MD PhD
Dep. of Orthopaedics, Regional Hospital West Jutland
Opponents:
Thomas Jakobsen, MD PhD DMSc
Dep. of Orthopaedics, Aalborg University Hospital
Ignacio Sanpera Trigueros, MD PhD
Dep. of Orthopaedics, University Hospital Son Espases, Palma
Bone bridge formation due to physeal injury may cause growth disturbances in children. However, not all physeal injuries cause bone bridge formation. The current treatment options for bone bridges are ineffective and the effect of intramedullary nailing and removal on axial growth is unknown.
The physeal healing upon grafting in a standardized gap was investigated in addition to effect of intramedullary nailing through an open femoral physis. All studies were carried out in an experimental juvenile porcine model.
The main findings were that fat grafts were ineffective in preventing bone bridge formation, whilst
autologous and allogenic cartilage were able to prevent bone bridge formation. Moreover, insertion and later removal of a retrograde femoral nail through the physis did not affect axial growth in the juvenile porcine model.
The perspectives are that future research should be focused on long-term studies, which will be performed.